mislim da stvari stoje ovako:

da bi se joni/koloidi srebra vezali za jone/koloide hlora treba im odredjena kolicina energije. smatram da prosta energija koju imaju pomenute cestice a koja potice samo od naelektrisanja nije dovoljno da se pomenute cestice spoje ...
znaci, da ponovim, mislim da joni/koloidi srebra iz srebrne vode nemaju tu potrebnu energiju i da se ne desava nista.
mozda bi se nesto desavalo kada bi se vode spojile i mesecima stajale zajedno ali ni to nisam siguran...
stajanje na suncu ili muckanje bi takodje mozda moglo da izazove pravljenje nezeljenog srebro-hlorida ali mislim da bi kolicine stvorene na ovaj nacin bile zanemarljive... i samim tim neskodljive, uglavnom mnogo neskodljivije od ostalih stvari kojima smo okruzeni!
sad sam u toku ovog pisanja poceo da se smejem...
jer sve ostalo sto unosimo u sebe, pre svega vazduh, zatim vodu, pa hranu je vec toliko zagadjeno da malo srebro-hlorida nas muljeviti stomak nece ni da primeti

pozdrav

Ok, definitivno je lakse onima koji se razumeju malo vise u hemiskim procesima , ali ja sam totalno zbunjena! Cackala sam malo po internetu i dosla do miliun stranica i svaka od njih govori nesto drugacije o srebrnu vodu. Jedni kazu da joniziracko srebro ne valja, drigu kazu da samo joni ustvari deluju, i sta ja znam... izgleda da svi se trude prodati svoju proizvod koji je najbolji! , Samo mi nisu jasne one stranice koje ne prodaju nista, jednostavno su resili obavestiti ljude o pravoj istini. Jedini problem je u tome da i takvih strana ima puno i sve velikodusno pricaju razlicitih stvari!

Procitala sam tu negde na ovim stranama da ne treba piti ili jesti nista nekih 20 min pred i posle uzimaje koloidnu vodu. Ok, znaci ipak trudimo se da sprecimo neku hemisku reakciju. A gde je ustvari problem; u tome da ako se srebro veze za neki drugi element postalo bi neaktivno, ili mozda ono deluje i dalje samo ima opasnost od talozenja u organizmu?

Naisla sam na jednu stranu http://cerebrex.com/silver.html
Nisam znala da prodaju vakve stvari, sta vi mislite o ovome?
Pozdrav svima

Mislim da ljudi puno vole filozofirati kod mnogih stvari.

Sve je jako slično, ali vide stvar iz svoje perspektive i drže se tog viđenja kao najboljeg i jedinog mogućeg.

Poslije svih ovih godina, mogu reći sljedeće:

- Radim kod kuće koloidno i ne zamaram se niti koncentracijama, niti jakosti struje i napona... napravim, pa kako bude. Niti jednom nije ispalo neupotrebljivo, niti štetno. Stvar je samo u koncentraciji - ako je otopina jača, odnosno veće koncentracije, treba ti manja količina za isti učinak kao i nekoncentrirana otopina. Jaka stvar. Litra destilirane je 5 kn.

- Primjetio sam da u zadnje vrijeme skupo prodaju mašine za pranje veša sa "srebrnim rešetkama" koje kao pročišćavaju vodu, ubijaju bakterije u vešu, odnosno dezinficiraju veš... haha... skupo prodaju ovu pamet koju tu imamo besplatno na forumu ( i na drugim mjestima na internetu )

- Zubar je prijatelju poslije nekakvog zahvata na zubu stavio 3 srebrne iglice oko zuba i rekao da su ove puno skuplje od onih konvencionalnih, ali ove istovremeno i dezinficiraju meso i nedaju da se zub zagnoji ili naotekne zbog infekcije. Zubar je bio dobar prijatelj, pa mu nije ništa više naplatio, ali drugima taj tretman dobrano naplaćuje.

- U bolnici u kojoj mi radi jedan drugi prijatelj, koji baš radi na zavijanjima i previjanjima, kaže da imaju specijalne zavoje, koji imaju neki srebrni kraj ( foliju ili prašak u kojem je srebro ) i koji se stavi na ranu, tako da se rana ne inficira. I naravno, to samo u specijalnim uvjetima, jer su kao jako skupi i kako zdravstvo nema love, jako rijetko se primjenjuju.

Ima i još drugih primjera, ali ovi osnovni daju pametnom čovjeku već dovoljno rezultata... samo malo treba promisliti i istražiti razne slučajeve sa širom percepcijom - i fenomenalni zaključci su tu !!!

U vezi zashto se posle CS ne jede...

Znaci uvjek kad se uzima neki biljni preparat, ili eto neshto kao shto je Cs koje treba da se upije, najbolje je da se uzima na prazan zeludnik i 30 minuta posle se ne jede, zato shto ako dodash hranu ili neka tecnost, onda oteznato je upijanje.. A cak moze da se desi da se hrana u zeludnik i pokvari, Shto je cesti slucaj. To se desi kad se ne jede pravilno, i ljudi urade neku ludu kombinaciju kao kebapcici i pomfrit, ili hamburger, a takva hrana se uvjek, al uvjek pokvari a s time i CS ili neki drugi bilni preparat koji mu doge kao propratna shteta...

Retko ko moze da se pohvali da uvjek jede pravilne kombinacije u svoje dnevne porcije tako da je najbolje da se pridrzavamo na uputstvo a to je da se uzima na gladan zeludnik i posle se ne jede 30 minuta...

Pozdrav

Iz vaseg poduzeg razgovora o koloidnom srebru ja sam zakljucio sledece:



Imam dva srebrna stapica 7cm duzine, 7mm sirine i jedan milimetar debjine. Zaronio bih ih po 5cm. Znaci da je povrsina jedne 8 cm2. Zaci 8 puta 0, 15 mA je oko 1mA (1,2).

Namera mi je da nabavim transformator sa 220V na 30V (pa da kasnije otpornicima smanjim voltazu na 24V ili manje), a da amperazu odrzavam oko 1mA (pritom sve delove da mi ortak, koji je elektrotehnicar, zbudzi u kutiju tj od toga napravi uredjaj koji ce da pusta jednosmernu struju od manje od 30V i od 1mA).

Da li je to optimalna struja za moju srebrnu elektrodu od 8cm2 (kada kazem povrsina mislim na obe strane plus obe povrsine debljine)?

lusha, dobrodošao!

Pa 1 mA je ok, ali će ti struja ovisiti o otporu ( razmaku elektroda, temperaturi destilirane vode, površini uronjenog dijela... ). Isto tako, ta struja raste kako se odvija postupak, jer je otopina sve vodljivija i vodljivija ( više srebrnih iona u njoj ).
Ako možeš regulirati napon, tako da struja bude par mA, to je već super rezultat. Dakle, stabilizator napona kod kojeg se napon mijenja kontroliranjem struje između elektroda. Što je struja manja, to su finiji komadići, što je poželjno, ali i proces dulje traje.

Puno uspjeha!

Peperutka,

Laser mozhesh nabaviti na svakoj pijaci (BitPazar e dobra opcija). Trazhish tezgu sa baterijama, svetiljkama i sl., i pitash za one male laserchice, koji su pre neke godine bili strashno u modi kod dece. Koshtaju 50-70 den (oko 1 euro).
Ko se igra sa srebrom, mora i da se zbuni. Nije to nishta. Kao i za sve istinite stvari, prochitachesh brdo suprotstavljenih tvrdnji. Onda ostaje na tebi da reshish s kojom filozofijom cesh da vozish dalje... Ne mresh znat, dok ne probash na sebi. Pa i tada, pripazi na to da nekad lekovite stvari imaju nezgodan uchinak (vidi onaj OPO-ov Herxhajmerov, ili kako beshe, efekt).

HEJ, a gde je taj post? Gde su OPO-vi postovi? Shta se tu zbiva?!?

HVALA!


Sve sam to skapirao, samo mi malo nije jasan odnos izmedju amperaze i napona... i samo sam pitao da li za elektrodu od 8cm2 (toliko je uronjeno) ok 1 mA. Ali skoo mi je kristalno jasno...... hvala svima na ovoj super temi na ovom super furumu.... jer da nije bilo vas ko zna gde i koliko dugo bih trazio informacije u vezi sa pravljenjem srebrne vode...

The Colloidal Silver Database Website
Summary of Toxicological Data of Certain Food Additives: Silver
WHO Food Additives Series 12
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Author: International Promgramme on Chemical Safety, World Health Organization
Original Source Format: http://www.inchem.org/documents/jecfa/j ... 12je19.htm
Copyrights: No Copyright Displayed
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Commentary
The following document, compiled by the World Health Organization, deals exclusively with stabilized silver compounds ( silver salts and proteins ) many of which have been known to be toxic for quite some time.
The reader will notice that many of the more extreme experimental data was conducted with rats ( and other animals ) and large amounts of highly concentrated silver compounds. It should be a simple matter to reproduce similiar experiments with colloidal silver produced via the electrolysis method to demonstrate the difference between product types.
In the world of science and medicine, as some of our included comments will show, careful scrutiny must be performed to weed out political slants from otherwise useful data.
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Silver does not occur regularly in animal and human tissues but is present in man's environment in air, water, soils and food as well as in specific products. In some marine species silver tends to accumulate in soft tissue. The shells and soft tissues of approximately 50 oysters (Crassostrea virginica Gmelin) analysed were for silver and other elements. The oysters were collected from 10 stations of various salinity ranges along the Georgia coast. Analysis was carried out by atomic absorption spectrophotometrically. The precision of the analysis was about ±5. Silver was below detectability in the shells (i.e. below 1 ppm) while the soft tissues was 28-82 (±10-20) ppm (Casarett and Doull, 1975; Windom and Smith, 1972).
Silver can be absorbed by the gastrointestinal tract. Retention is apparently greatest in the reticulo-endothelial organs. After intravenous injection the concentrations were present in decreasing order in spleen, liver, bone marrow, lungs, muscle and skin (Browning, 1969).
Various studies and clinical observations indicate that silver salts can be absorbed from the lungs, gastrointestinal tract and such insured epithelia as nasal mucosa, conjunctiva, and skin. Absorbed silver is then stored in the reticulo-endothelial cells of the skin, mucous membranes, liver, spleen, possibly bone marrow, in basement membranes, especially those of the renal glomerulus, and presumably in muscles (Ham and Tangue, 1972; Kanai et al., 1976; Bader, 1966; Anderson, 1966; Voldrich et al., 1975).
Radiosilver (110mAg) administration to mice, rats, monkeys and dogs by oral intravenous and intraperitoneal routes was excreted for more than 90% in the faeces, 90% or more of oral doses were not absorbed. Whole body retention in mice, rats and monkeys was less than 1% of the initial dose after one week. In the same period less than 10% was retained in dogs (Fnrchner et al., 1968).
The major route of excretion is via the gastrointestinal tract, predominantly through desquamation of silver containing cells of the alimentary tract. Urinary excretion has not been reported to occur even after intravenous injection (Casarett and Doull, 1975; Kent and Mc Cance, 1941) It seems that even mild degrees of liver damage considerably impair the ability of the liver to excrete quite small doses of silver (Petering, 1976). Unlike lead or mercury there is no evidence that silver is a cumulative poison (Petering, 1976).
No information was obtained on the biotransformation of silver in the animal body except that absorbed ionic silver is transformed into metallic while being deposited in tissues (Petering, 1976)
Numerous enzymes were inhibited in vitro by silver ions. High affinity to sulfhydryl and histidine imidazole groups was observed. Silver ions compete with molecular oxygen as a hydrogen acceptor, resulting in the inhibition of glucose oxydase (Nakamura and Ogura, 1968)
Protargol, a silver-protein complex containing 8% silver inhibited the in vitro prostaglandin E2 synthesis by bull geminal vesicles even at concentrations of 10-7M (Deby et al., 1973).
Glutathione peroxydase activity in the liver of rats treated with 76 and 751 ppm silver (as silver acetate) for seven weeks was respectively 30% and 4% of the control values (Swanson et al., 1974).
After a single s.c. injection (3 mg silver/kg bw) AgNO3 induced the synthesis of a low molecular weight protein in the liver of rats, with the characteristics of metallothionein induced by cadmium, zinc or mercury salts (Winge et al., 1975).
Silver ion is a very toxic substance when viewed from the standpoint of its action of an inhibitor of enzymes and as a metabolic inhibitor of lower forms of life. Biochemically, the silver ion (Ag+) can act as potent enzyme inhibitor (Chambers et al., 1974). It has been reported (Wagner et al., 1975) that in vitro administration of silver dramatically decreased liver glutathione peroxidase in rats fed Se-supplemented diets with or without vitamin E. It seems therefore that silver acetate exerts its antagonistic effects on Se (silver induces Se deficiency signs) through an effect on the activity of biosynthesis of glutathione peroxidase
Much of the biologic action of silver can be attributed to the reaction of silver ion with sulfhydryl groups to produce stable silver mercaptide (Petering, 1976).
Cooper and Jolly (1970) in a review of the ecologic effects of silver have pointed out that the current experimental practice of seeding clouds with silver iodide to promote rainfall may lead to new hazards for both man and natural biologic systems if the practice is extended (Petering, 1976).
TOXICOLOGICAL STUDIES
Special studies on carcinogenicity
Sarcomas, malignant fibrosarcomas, fibromas, fibro-adenomas and invasions of muscle with corrective tissue were observed after implantation of foil, platelets and pellets made of silver or dental
alloy under the skin of mice and rats (Oppenheimer et al., 1956; Shubik and Hartwell, 1969)
Special studies on mutagenicity
No DNA damaging capacity was observed in a recombination-assay with AgCl in a Bacillus Subtilis strain (Nishioka, 1975).
Acute toxicity studies
Oral administration of 50 mg AgNO3/kg bw to mice caused death in 50% of the animals in a 14 day observation period (Goldberg et al., 1949).
Intraperitoneal administration of 2 ml of an aqueous solution containing 0.239 M AgNO3 to guinea pigs (0.216 g AgNo3/kg bw) was fatal in 6/10 animals after seven days (Wahlberg, 1965).
Intraperitoneal injection of 20 mg AgNo3/kg bw in rabbits caused death accompanied by degeneration of liver parenchyma and kidney tubules. Silver granules were observed in these organs (La Torraca, 1962).
Subcutaneous injection of 7 mg AgNO3/kg bw to rats affected testis histology and spermatogenesis. After 18 hours the peripheral tubules were affected and some central tubules were completely degenerated. Some tubules recovered but not the duct system (Hoey, 1966).
A single dose of 500 mg of colloidal silver was lethal to dogs in 12 hours (Shouse and Whipple, 1931). Prior to death there was anorexia, weakness, loss of weight, and anaemia. Death was due to pulmonary congestion and oedema.
Short-term studies
Rats
Rats (90-100 g) were given a 0.25% solution of AgNO3 in distilled water as drinking water for a period ranging from 1 to 12 weeks. Rats were killed at 1, 2, 3, 4, 8 and 12 weeks and at 1, 2, 3, 6, 10 months and also 16 months after silver administration had stopped. Deposition of silver in the glomerular basement membrane was noticed one week after the initiation of treatment electron microscopically (Ham and Tange, 1972).
1500 ppm Ag1 (as acetate) in drinking water for two to four weeks caused liver necrosis and death in vitamin E deficient rats. The effect was prevented by 120 ppm D- -tocophirylacetate and partially by 1 ppm Se (Diplock et al., 1967).
Addition of silver acetate to the diet (130-1000 ppm) or drinking water (1500 ppm) of weaning rats fed a vitamin E deficient diet, precipitated a rapidly fatal hepatocellular necrosis and muscular dystrophy on day 14 of the treatment or subsequently. No changes were observed in liver of rats given silver acetate and vitamin E supplements. The mitochondrial changes possessed some of the features seen in rats fed a diet deficient in vitamin E and selenium. A reduced availability of selenium by silver in vitamin E deficient rats is postulated (Grasso et al., 1969).
Rats fed a casein-based diet were given 0.76 and 751 ppm silver (as acetate) in drinking water for a period of seven weeks. Dietary Se (0.5 ppm as Na2SeO3) prevented growth depression observed in rats receiving 76 ppm silver and markedly improved growth and survival of those given 751 ppm, but increased liver and kidney silver levels.
Liver glutathione peroxidase activity of the treated groups supplemented with selenium was respectively 30% and 4% of the controls. Glutathione peroxidase of erythrocytes was not affected (Swanson et al., 1974).
Cyanocabalamine (3 ppm), vitamin E and selenium (0.05 and 1 ppm) were found to antagonize silver-induced liver necrosis in rats (Bunyan et al., 1968).
Rats (six per group) were treated with drinking water containing 0.5, 2 and 20 mg Ag/l for 6-12 months. 2 mg Ag+/l decreased the nucleic acid level in brain and liver after one year and 20 mg Ag+/l increased RNA and DNA contents of the brain after six months and caused dystrophic changes in the brain accompanied by a decrease in
nucleic acid level after 12 months. The liver was less sensitive towards silver than the brain (Kharchenko et al., 1973).
Groups of eight rabbits received 0, 0.00025, 0.0023, 0.025 and 0.25 mg Ag/kg via their drinking water during 11 months. Marked effects on immunological capacity (measured as phagocytosis) and histopathological changes of nervous, vascular and glial tissue of the encephalon and medulla were observed in the groups receiving 0.025 and 0.25 mg Ag/kg bw. Treatment had no effects on haemoglobin, R.B.C., differential W.B.C., proteinogenic function of the liver and serum SH groups. Rats treated with same amounts of silver showed affected conditioned reflexes (Barkov and El piner, 1968).
Groups of 20 chicks received 0, 10, 25, 50, 100 and 200 ppm silver during four weeks in combination with 0, 10 or 25 ppm copper in the diet. Silver at 100 ppm reduced growth in the copper deficient but not in the control chicks. At 50 ppm mortality was increased in the copper deficient group, but not in those receiving copper. 10 ppm silver reduced the haemoglobin concentration and the elastin content in the aorta in deficient chicks. These effects were completely overcome by the addition of copper to the diet (Hill and Matrone, 1970).
Turkey poults given dietary silver (900 ppm of added silver nitrate) exhibited reduced body weight gain, haemoglobin, packed cell volume, and aortic elastin content, as well as significantly increased ratio of wet heart weight to body weight. The enlarged hearts were attributed to a copper deficiency induced by the dietary silver. Adding extra copper offset the silver-induced condition (Peterson et al., 1973; Jensen et al., 1974),
OBSERVATION IN MAN
Absorption of silver resembles whole body retention. It is retained in all body tissues (Hamilton et al., 1972a; Tripton et al., 1966). The silver content of the miocardium, aorta and pancreas tends to decrease with age (Bala et al., 1969) although the amount of silver in the body increases with age (Hill and Pillsbury, 1939). The concentration of silver in healthy human tissues from the United Kingdom was 1-9 µg/kg ash was found. The average silver contents in wet tissue of normal Americans was about 0.05 µg/kg (Tripton, 1963).
The intake from the diet is estimated at 27 µg/day (Hamilton and Minski, 1972) up to 88 µg/day (Kehoe et al., 1940).
Silver toxicity is manifested in a variety of forms, some proven others suspected. Proven forms include: argyria, gastrointestinal irritation, renal and pulmonary lesions. Suspected forms include, among others (ill-defined) arteriosclerosis (Casarett and Doull, 1975).
Argyria denotes the slate blue colour observed in parts of the body of persons exposed chronically to silver (Anderson, 1966). Epidemiologically, two types of argyria are recognized: industrial argyria and iatrogenic argyria.
Regardless of type there are two forms of argyria, local and generalized. The local form involves the formation of grey blue patches on the skin or may manifest itself in the conjunctiva of the eye. In generalized argyria the skin shows widespread pigmentation, often spreading from the face to most uncovered parts of the body. In some cases the skin may become black with a metallic lustre. Heavy pigmentation of the eye structures can interfere with vision (Casarett and Doull, 1975). Except for this adverse effect argyria is solely a cosmetic problem. The slate blue colour of argyria is not entirely due as one might suspect, to the deposition of metallic silver (Petering, 1976), but largely to an increased deposition of melanin. Silver has a melanocyte-stimulating property (Rich et al., 1972). Cases of generalized argyria have occurred after ingestion or chronic medicinal application of gram quantities of silver. Silver was absorbed during prolonged (nine months) nasal application of Targesine (silver solution). It was calculated that during this time 7000 ml of solution containing 210 g silver had been used (Voldrich et al., 1975)
After chronic medical and occupational exposure to silver, argyria and argyrosis are the most common findings. Although intravenous administration of a total of 0.91-7.6 g (average 2.39) silver as silver arsphenamine in a period of two to nine years has caused argyria, hundreds of patients have received up to 1.7 g Ag (as arsphenamine) without developing argyria.
In argyria silver is regularly deposited in blood vessels, connective tissue, skin, glomeruli of the kidney, choroid plexus, mesenteric glands and thyroid. Adrenals, lungs, dura mater, bones, cartilage muscle and nervous tissue are minimally involved as deposition sites for silver.
In workers argyrosis of the cornea may be accompanied by turbidity of the anterior lens capsule and disturbance of the dark adaptation, usually not resulting in loss of vision.
Argyria is observed only in connexion with occupational medical exposure or after cosmetic application of silver (Hill and Pillsbury, 1939).
The systemic effects of silver are not extensive because of the poor absorption of silver compounds from the intestinal tract (Petering, 1976). It is considered that 10 g of silver nitrate taken orally is a lethal dose of man, although recovery from smaller doses has been reported (Cooper and Jolly, 1970). The systemic effects of a
lethal dose are preceded by severe haemorrhagic gastroenteritis and shock. According to Goodman and Gilman (1965) the silver ion seems first to stimulate and then depress structures in the brain stem. Central vasomotor stimulation results in a rise in blood pressure. At the same time there is bradycardia due to central vagal stimulation. Death eventually results from respiratory depression.
REFERENCES
Anderson, W. A.D. (1966) In: Pathology, C. V. Mosby, Saint Louis, 1, 73
Bader, K. F. (1966) Organ deposition of silver following silver nitrate therapy of burns, Food and Cosmetics Toxicol., 5, 435
Bala, Yu., Lifshits, V. M., Plotko, S. A., Aksenov, G.I. and Kopylova, L.M. (1969) Age levels of trace elements in the human body, Voroneah, Gos. Med. Inst., 64, 37-44, cited by Carson and Smith, 1975
Barkov, G. D. and El 'piner, L. I. (1968) The need for limiting the silver content of drinkingwater, Gigiena i Sanit., 33, 16-21
Browning, E. (1969) Toxicity of industrial metals, 2nd ed. Butterworths, London, 296-301
Bunyan, J., Diplock, A. I., Cawthorne, M. A. and Green, J. (1968) Vitamin E and stress, VIII. Nutritional effects of dietary stress with silver in Vitamin E-deficient chicks and rats, Brit. J. Nutr., 22, 165-182
Casarett, L. J. and Doull, J. (1975) In: Toxicology the basic science of poisons, MacMillan, New York, pp. 967-969
Chambers, J., Krieger, C. G., Kay, L. and Stroud, R. (1974) Silver ion inhibition of serine proteases: Crystallographic study of silver- trypsin, Biochem. and Biophys. Res. Comm., 59, 70-74
Cooper, C. F. and Jolly. W. C. (1970) Ecological effects of silver iodide and other weather modification agents; A review, Water Resources Research, 6, 88-98
Deby, C., Bacq, Z. M. and Simon, D. (1973) In vitro inhibition of the biosynthesis of a prostaglandin by gold and silver, Biochem. Pharmacol., 22, 3141-3143
Diplock, A. T., Green, J., Bunyan, J,, McHale, D. and Muthy, I. R. (1967) Brit. J. Nutr., 21, 115
Furchner, J. E., Richmond, C. R. and Drake, G. A. (1968) Comparative metabolism of radionuclides in mammals. IV. Retention of silver 110-m in the mouse, rat, monkey and dog, Health Phys., 15, 505-514
Goldberg, A. A., Shapero, M. and Wilder, E. (1949) Antibacterial colloidal electrolytes: the potentiation of the activities of mercuric-, phenylmercuric- and silver ions by a colloidal sulphonic anion, J. Pharmac. Pharmacol., 2, 20
Goodman, L. S. and Gilman, A. (1965) In: The pharmacological basis of therapeutics, MacMillan, New York, 3rd ed., p. 965
Grasso, P., Abraham, R., Handy, R., Diplock, A. T., Goldberg, L. and Green, J. (1969) The role of dietary silver in the production of liver necrosis in Vitamin E-deficient rats, Exp. Mol. Pathol., 11,
186-199
Ham, K. N. and Tange, J. D. (1972) Silver deposition in rat glomerular basement membrane, Aust. J. Biol. Med. Sci., 50, 423-434
Hamilton, E. I., Minski, M. J. and Cleary, J. J. (1972) The concentration and distribution of some stable elements in healthy human tissues from the United Kingdom, Sci. Total Environ., 1,
341-374
Hamilton, E. I. and Minski, M. J. (1972) Abundance of chemical elements in man's diet and possible relations with environmental factors, Sci. Total Environ., 1, 375-394
Hill, C. H. and Matrone, G. (1970) Chemical parameters in the study of in vivo and in vitro interactions of transition elements, Fed. Proc., 29, 1474-1481
Hill, W. R. and Pillsbury, D. M. (1939) Argyria, the pharmacology of silver, The Williams and Wilkins Co., Baltimore, Maryland, cited by Carson and Smith, 1975
Hoey, M. J. (1966) The effects of metallic salts on the histology and functioning of the rat testis, J. Reprod. Fort., 12, 461-472
Jensen, L. S., Peterson, R. P. and Falen, L. (1974) Inducement of enlarged hearts and muscular dystrophy in turkey poults with dietary silver, Poult. Sci., 53, 57-64
Kanai, A., Yamaguchi, T. and Nakajima, A. (1976) The analytical electron microscopic study of the corneal and conjunctival deposits of pigments and other substances, Part 2: Conjunctival argyrosis,
Acta Soc. Ophthalmol. Jpn., 80, 385-389
Kent,

Ovo nam je poslano na mail, da razmislimo želimo li i dalje koristiti CS. Molim komentar stručne osobe ili nekoga tko se smatra dovoljno stručnim prokomentirati ovaj tekst. Moram priznati da me malo pokolebao, iako praktična primjena CS-a pobija činjenice iz teksta. Koristimo ga duže no što tamo piše da je dovoljno za sve te komplikacije, pa ne vidimo neke prevelike probleme i komplikacije u organizmu, izuzev eventualno prob. smetnji, koje kratko traju...ili link sa pozitivnim mišljenjem nekoga iz branše tradicionalne medicine, farmacije i sl.
I isprika što sam prebacila i neke nepotrebne podatke, npr. na kraju..pozdrav

Prvo, antibiotici su steti, pa niko ne pravi halabuko oko toga- jer je to marketing. Drugo, marketing svetske frmaceutske industrije (koja najvise ulaze u duvansku industriju) se trudi da spreci konzumiranje bilo cega sto nije u njihovom zatvorenom krugu trovanja- lecenja.

Inace nisam procitao tekst, procitacu ga, ali ovo je univerzalno.

I shta je onda problem sa ovim tekstom? Kad se pazhljivije chita, sve je u redu i u saglasnosti sa drugim stvarima koje smo chitali. Fino pishe na pochetku, a i dalje u tekstu, da su sva istrazhivanja pravljena na razne srebrne SOLI (AgCl, AgNO3 i sl., za koje vec imamo informacija koliko su toksichne) i srebrne proteine, za koje sam chitao slichne stvari. Ondak drugo, josh interesantnije... Ajmo prorachunati kolike kolichine srebra inache chovek koji konzumira srebro unosi u organizam. Ako srebro pravi problizhno 5ppm, to bi bilo priblizhno 5mg Ag, po litri koloidnog srebra. Znachi, kad bi u jedan dan popio celu litru srebrne vodice, ondak bi u organizam uneo celih 5mg srebra, ili nekih 0.1-0.05mgAg/kgbw (miligrama srebra po kilogramu telesne tezhine) za osobe od 50 do 100 kila tezhine. Sad opet prochitajte podatke o onim jadnim pacovima, kucama i macama kojima su injektirali po 20-50mg srebra (u stvari srebro-nitrata, ali hajde), po kilogramu telesne tezhine, na dan, shto bi u prevodu znachilo da chovek od 100kg treba popiti 1000 (hiljadu) litara srebrne vode od 5ppm, na dan, da bi sebe ubio na tako spektakularan laboratorijski nachin! A poshto mi pijemo samo po nekoliko gutljaja na dan, onda u telu unosimo valjda nekih 0.01-0.005mgAG, t.j. 5-10µg na dan. Za usporedbu: (citat) "The intake from the diet is estimated at 27 µg/day (Hamilton and Minski, 1972) up to 88 µg/day (Kehoe et al., 1940)."

And that's all I have to say about that...

P.S. Nego oce li neko da gukne gde su OPO-vi komentari? Ako ih je OPO povukao, tuzhit cu ga! Pa nemozhe tako! Neshto lepo davati, pa ondak uzeti... Ne, stvarno, shta se zbiva?

Sada sam u dilemi !!!
Mogu trenutno da nabavim srebro chistoce 999,5 tj. 99,95%, medjutim po rechima zlatara koji mi nabavlja to srebro ono je "rafinisano" tj dobijeno hemijskim putem. Takodje mi reche da meni treba elektrolizno srebro a ono je chistoce 99,99%. U onom prvom se navodno mogu naci i tragovi nekih kiselini ili neshto slichno.
Da li da uzimam 99,95 ili da josh malo sachekam 99,99?

Molim vas komentar na ovo!

Hvala!
PS! Za Rasa!
Gde nabavljash srebro za sebe i koje je chistoce???

Pa bolje je što čišće...

ovisi od čega su primjese ovih 0,05% nečistoće ( nije svejedno da li je to npr. bakar ili arsen... )



Sad lijepo uzmite bilo koje tablete i pročitajte štetne i nus-pojave kod tih tableta. Popis će biti barem 2-3 puta duži od ovoga.

Isto, slažem se sa joshneznamsvojeime, ovi testiraju sa srebrnim solima o kojima smo već govorili da nisu baš poželjne. Nije isto srebro u spoju ili čisto srebro. Ove jadne životinje su dobile količinu SOLI, a ne koloidnog srebra ( kako se tvrdi u tekstu ).

Isto tako ako popijete npr. 3 tablete nekog antibiotika dnevno - nije isto kao ida ih popijete 300 ili 3000 ( koliko puta više daju životinjama po kg ).

Ako djeluje ( ili ne djeluje ) nešto na životinje - to nemora značiti da će i na ljude, i obratno. Svaka vrsta ima svoje karakteristike podložnosti na neke supstance, ali i jedinke unutar te iste vrste.

Po tim spisima bih ja trebao već odavno biti mrtav?!? Ili možda bar toliko siv da bi me zamjenili za Sivog kad bi me vidjeli na ulici... hehe!

da li je neko od vas koristio srebrnu žicu koju koriste za varenje električari tj da li imate kakvu informaciju o tome i ako može gdje se može nabaviti u Hrvatskoj ili Srbiji

http://www.h-minus-ion.org/water-tx-device-1.html


Ovo nije koloidno srebro, ali voda jeste. Covek nudi ( izgleda veoma jednostavno ) nacrt, a dao je i na slobodno koristenje svoj identifikacioni broj za ostvarivanje popusta za nabavu praha poludragog kamena a ostalo po principu uradi sam!!!

Ko se zanima za ORBUS elemente neka pogleda.



Iskreno , ja se ne razumem, mislim da bi ovo nekima moglo biti zanimljivo.

Silver ion is a very toxic substance when viewed from the standpoint of its action of an inhibitor of enzymes and as a metabolic inhibitor of lower forms of life. Biochemically, the silver ion (Ag+) can act as potent enzyme inhibitor (Chambers et al., 1974). It has been reported (Wagner et al., 1975) that in vitro administration of silver dramatically decreased liver glutathione peroxidase in rats fed Se-supplemented diets with or without vitamin E. It seems therefore that silver acetate exerts its antagonistic effects on Se (silver induces Se deficiency signs) through an effect on the activity of biosynthesis of glutathione peroxidase
Much of the biologic action of silver can be attributed to the reaction of silver ion with sulfhydryl groups to produce stable silver mercaptide (Petering, 1976).

Kako se kol. srebro ponasa prema enzimima???
Nije mi samo jasno.... da li je moguce da se kol. srebro u nasem organizmu spoji u srebrno nitrat ili nesto slicno otrovno???


Mene muci problem povlacenja desni.... da li srebro moze da utice na to???? (ubija bakterije, ali da li ako stalno sprecavam bakterije da mi nagrizaju desni ce mi se desni vratiti u normalu?????)
Ko da sam kod stomatologa.....

Probaj. Naštetiti ne može... ako je povlačenje desni vezano uz neke bakterije ( koje tu nebi trebale biti ) onda će vjerojatno rješiti... možda ne za dan-dva... ali možda kroz neko vrijeme... tjedan, mjesec... teško je točno odrediti.

Prvo pozdrav svima na forumu!
A sad pitanje.koja kolicina srebra(otprilike), se unosi ako se koristi srebrini escajg,recimo za dva obroka u toku dana?
Jeli ulazi u one okvire preporucene doze?

Evo i ja napravih prvu kolichinu srebrne vodice !!!
-Pochetna struja mi je bila 0,30 - 0,35 mA
-Elektrode - " Shtapici" 2mm prechnika uronjeni oko 7 cm u vodu razmaka oko 3-4 cm
vreme trajanja 2 sata i 30 min, napon 12 V , struja na kraju 1,05 mA.
Chistoca srebra je 99,5%.
Kada sam koristio kalkulator koji je na forumu dobijao sam razlichite rezultate u zavisnosti da li stavljam zarez ili tachku - kako za amperazu tako i za vreme. Nisam primetio neshto bash metalan ukus.
-Elektrode su blago pocrnele ( i jedna i druga jer sam menjao polaritet u toku procesa ) ali sam ih lagano obrisao papirnom maramicom . Josh da napomenem da sam vodu meshao u toku procesa plastichnom kashicicom.
-Deshavalo se (redovno) da struja padne prilikom meshanja.
-nije bilo izmaglice ali je bilo par mehurica na kraju + elektrode.
Josh samo nesto, a to je pojava nekog taloga (u tragovima) svetle boje - inache voda je bistra kao i na pochetku procesa.

Neka mi neko iskusniji kaze shta i koliko sam dobio i kako se koristi kalkulator ( da li tachke ili zarezi ).

HVALA!!!!